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B Bacillary Angiomatosis Etiology : Bartonella henselae or Bartonella quintana + CD4 < 100; transmitted by cats or lice Appearance : erythematous to violaceous papules that grow into nodules; very friable and bleeds profusely At risk : HIV, immunosuppressed, organ transplant recipients Tx : erythromycin or doxy Bacterial Folliculitis Etiology : S. aureus , unless it is Gram negative (Escherichia coli, Pseudomonas aeruginosa, Serratia marcescens, Klebsiella or Proteus species ) or Hot tub Folliculitis (Pseudomonas aeruginosa) Appearance : follicular pustules, erythematous nodules Tx : warm compress, anti-inflammatories, mupirocin Basal Cell Carcinoma ***MC skin cancer Etiology : UV radiation, mutation in patched (PTCH) tumor suppressor gene Appearance : skin colored or pink pearly papule with rolled borders and telangiectasias Dermoscopy : sharp demarcated dark vessels Tx : excision, MOHs, cry o, PDT Behçet Disease Etiology : unknown, assumed to be connected to autoimmune response; associated with HLA-B51 Appearance : oral + genital ulcers that are 3-5 mm, round to oval ulcers with peripheral rim of erythema + yellowish adherent exudate centrally (indistinguishable from aphthous ulcers) At risk : eastern and central Asian, Mediterranean; 30-40 yo Blastomycosis Etiology : inhale spores of Blastomyces dermatitidis (found in wood and soil, on dogs) Appearance : purplish-gray verrucous lesions with heaped borders or friable lesions that ulcerate Other sx : flu-like sx, productive cough Location : face, neck, extremities At risk : south/central and mid-western America, immunocompromised, HIV Tx : itraconazole,amp B for severe disease 1/5 Blue Nevus Etiology : incomplete migration of melanocytes from neural crest to the skin during fetal development Appearance : dark blue macule, papule, or plaque Location : hands, feet, face At risk : women, asians Tx : if excised ("looks like a black hole" = super deep) Blueberry Muffin Syndrome Etiology : Rubella, CMV, tumors, blood disorders Appearance : descending blue, violaceous macule s + papules Ass. sx: anemia, hepatosplenomegaly, IUGR, jaundice Bowen disease / In situ SCC Etiology : UV radiation, HPV, arsenic exposure, immune suppression Appearance : irregular red/orange/brown scaly plaques Tx: cryo, observe, excise Bowenoid papulosis Etiology : HPV 16,18 Appearance : reddish-brown papules Location : anogenital region Tx: monitor, ED, laser, cryo 1/3 Bullous Diabeticorum Etiology : diabetes Location : lower extremities Tx : self resolving 1/4 Bullous Impetigo Etiology : S. aureus (exfoliative toxins A + B which targets desmoglein 1) Appearance : thin roofed bullae that tend to rupture spontaneously and ooze leaving a yellow crusty rim At risk : <2 yo Location : face, trunk, extremities, buttocks, perineal regions Tx: oral flucloxacillin Go to link Go to link Go to link Go to link Go to link Go to link Go to link Go to link Go to link 1/2 Bullous Pemphigoid Etiology : Autoantibodies to BP230 + BP180 (hemidesmosomes) Appearance : large, tense, fluid-filled blisters with urticarial/erythematous base Location : lower abdomen, upper thighs, or armpits; typically oral mucosa is spared Ass. with : HBV Tx: topical or systemic steroids Burns Etiology : any external heat/radiation source Types : - Superficial burn = localized, dry, blanching redness with no blisters - Superficial partial-thickness burn = blisters, blanches with pressure, swollen, warm - Deep partial-thickness burn = blisters that are easily unroofed, does not blanch with pressure and painful only to deep pressure - Full-thickness burn = white, waxy, dry, inelastic, leathery, does not blanch with pressure, painless - Deeper injury burn = white, dry, inelastic, does not blanch with pressure, painless Superficial burn Superficial burn Superficial burn Superficial partial-thickness burn Superficial partial-thickness burn Superficial partial-thickness burn Superficial partial-thickness burn Full-thickness burn Full-thickness burn Full thickness burn Full-thickness burn A B C D E F G H I J K L M N O P Q R S T U V W X Y Z

A Acanthosis Nigricans Etiology: insulin resistance Appearance : hyper pigmented leathery plaque Location : back of the neck, axilla, groin Associated with : gastric or lung adenocarcinoma, Cushing Syndrome, PCOS, diabetes, obesity Tx : treat underlying condition Acanthosis Palmaris / Tripe Palms Etiology: internal malignancy Appearance : thickened velvety pattern of skin often gives a yellow hue to the skin Location : palms and dorsal hands, soles of feet At risk : males Associated with : bullous pemphigoid, psoriasis Tx : treat malignancy Accessory Tragus Define : common, benign, congenital anomaly of the ear Appearance : small, skin-colored nodule Location : anterior to tragus Tx : excise Acne Keloidalis Nuchae Etiology: short hair cuts, friction from headgear or collars Appearance : flesh colored, dome shaped papules + pustules Sx : itchy Location : posterior scalp + neck At risk: African-Caribbean descent with dark curly hair Tx : change hairstyle, reduce friction, mild to moderate steroids with topical retinoids, oral doxy Acne Vulgaris Etiology : folliculosebaceous unit is blocked, swollen, ruptures, and inflames the surrounding skin Location : face, chest, upper back At risk : oily skin, teenagers Tx : tretinoin, adapalene, doxy, spironolactone, OC Ps , isotretinoin Types : comedones: open and closed papules pustules nodules Acrochordons / Skin tags Etiology: excess friction, insulin intolerance Appearance: skin colored pedunculated, fleshy papules Location : high friction areas like under breast, neck, axillae, groin Associated with : obesity, diabetes, pregnancy, acromegaly Tx: snipping, LN, ED Actinic Keratosis (AKs) Etiology: sun exposure Appearance: pink/hyperpigmented thin scaly plaque Location : sun exposed areas Prog : 10-15% turn into SCC Tx: cryo, 5-FU, imiquimod, PDT African Trypanosomiasis Etiology: T.b. rhodensiense, T.b. gambiense Appearance: - At site of Tsetse fly bite, chancre forms with enlarged lymph nodes - 2-3 weeks later, a central necrotic eschar forms - 6-8 weeks later, trypanids form (red patches, urticaria, targetoid lesions) Tx : before the meningoencephalitic phase = suramin; meningoencephalitic phase = melarsoprol 1/3 AL Amyloidosis Etiology: accumulation of an immunoglobulin light chain (lambda > kappa) protein Appearance: waxy skin with periorbital purpura (ex: Raccoon eyes) Tx : high dose plasma cell directed chemotherapy with melphalan and dexamethasone Albinism Etiology: mutation of OCA 1A, OCA 1B, and OCA 2 (oculocutaneous) = decreased tyrosine activity or defective tyrosine transportation Inheritance : AR Appearance: diffuse depigmentation Allergic Contact Dermatitis Etiology: type IV hypersensitivity reaction to allergen (nickel, poison ivy, PCN, detergents) Appearance: erythematous vesicular rash with edema Tx : find causative agent and avoid, emollients and topical steroids Go to link Go to link Go to link Go to link Go to link Go to link Go to link Go to link Go to link 1/2 Alopecia Areata Etiology : autoimmune condition Appearance : hairless, smooth patches without scale, erythema, or inflammation Test : positive hair test at periphery Ass. conditions : autoimmune disorders, Down syndrome , atopy Dermoscopy : exclamation point hairs Tx : topical clobetasol solution , minoxidil, intralesional triamcinolone Angioedema Etiology: increase in local capillary permeability, usually mediated by mast cells, histamine, or bradykinin release Appearance : swelling of dermis and SQ tissue Location : eyes, lips, genitals At risk : pts with chronic urticaria Sx : painful or burning, but not pruritic Tx : antihistamines for mild acute cases, oral prednisone for more severe cases, TXA or omalizumab for refractory recurrent angioedema Angiosarcoma Etiology : 20% have history of radiation to head or neck Appearance : blue or purple macular, sometimes raised or nodular; often become ulcerated or hemorrhagic Location : face + scalp Tx : complete resection with wide margins Prog : 5 yr survival is ~35% Angular Cheilitis Etiology: most commonly occurs due to prolonged exposure of the corners of the mouth to saliva and its digestive enzymes Appearance : erythematous fissuring, thin scales and crust Location : corner of mouth At risk : pts with poor health Ass. conditions: iron deficiency anemia, vitamin B deficiencies, protein malnutrition, chronic inflammatory diseases (IBD, Crohn Disease, Sjogren Syndrome) Tx : most cases resolve by itself, improve hydration, lip emollients 1/8 Aphthous Ulcer / Canker Sore Etiology : unknown antigen stimulates keratinocytes via increase pro-inflammatory cytokines Triggers : emotional stress, lack of sleep, mechanical trauma, nutritional deficiency, viral infections, certain foods or toothpastes Appearance : round to oval ulcer with peripheral rim of erythema + yellowish adherent exudate centrally Location: mucosa of lips, oral mucosa, tongue margins Tx : heals spontaneously, avoid triggers Aplasia cutis Etiology : intrauterine trauma, vascular compromise, infection (HSV, VZV), meds (methimazole), Patau syndrome (on occiput) Appearance : well-demarcated erosion or ulcer with loss of hair Tx : heal spontaneously Arsenical keratosis Define : development of thickened, wart-like growths on the skin due to chronic arsenic exposure Appearance : multiple, small, yellowish, and horny ( keratotic) papule s or plaques Location : palms + soles At risk : farmers, well water use Tx : cyro, ED, 5-FU, imiquimod, oral retinoids Arterial Ulcer Etiology : peripheral artery disease (atherosclerotic stenosis) Appearance : well defined punched out ulcer At risk : pts that smoke, have DM, high BP, high cholesterol, RF, RA, obesity Location : distal toes Tx : lifestyle changes, wound care Go to link Go to link Go to link Go to link Go to link Go to link Go to link Go to link Go to link 1/2 Atopic Dermatitis / Eczema Etiology : type 1 HS reaction associated with other atopic diseases; loss of function of FLG gene = mutation in filaggrin Appearance : irregular border pink plaque with scale +/- lichenification Location : flexural surfaces (adults) vs extensor surfaces (children) At risk : ‘atopic tendency’ clustering with hay fever, asthma, and food allergies Tx : avoid skin irritants, emollients, topical steroids, bleach baths, tacrolimus, Dupixent, Nemolizumab, and so much more Prog : 20% of children with AD had persistent sx 8 years later; children who developed AD before 2 yo had a lower risk of persistent sx than those that developed AD later in life Atopic Eruption of Pregnancy (AEP) / Prurigo of Pregnancy Etiology : pregnancy causing cytokine imbalance Appearance : hyper pigmented or erythematous papules that are often grouped together Location : extensor surfaces At risk : 2nd - 3rd trimester Prog : no increased risk to fetus, resolve after pregnancy Tx : topical steroids, benzoyl peroxide, emollients 1/5 A B C D E F G H I J K L M N O P Q R S T U V W X Y Z

C Café Au Lait Etiology : epidermal melanocytes have excessive numbers of melanosomes in that specific area Appearance : well-demarcated hyperpigmented oval or round shaped patch, pigment is evenly distributed Ass. with : Neurofibromatosis, McCune-Albright syndrome, Fanconi Anemia Tx : lasers Calciphylaxis Etiology: necrosis of skin + fatty tissue, typically in ESRD pts; can occur in those with high or normal levels of serum calcium + phosphate At risk : females, obesity, immunosuppressed Appearance : - begins as surface purple retiform purpura - then turns black in the center as a stellate shaped purpura - then turns into dry gangrene + ulcerates Tx : normalize calcium + phosphate levels associated with renal failure; IV infusions of sodium thiosulfate Go to link Go to link Go to link Go to link Go to link Go to link Go to link Go to link 1/2 Candidal Diaper Dermatitis Etiology : Candida albicans Appearance : erythematous plaques with peripheral scaling + satellite papules or pustules Locations : warm, moist areas Tx : clotrimazole cream Carbuncle Etiology : multiple bacterial folliculitis (furuncles/boils); typically S. aureus - mnemonic: "multiple furuncles fit in a CAR" Appearance : erythematous pustules surrounding a hair follicle Ass. with : Neurofibromatosis, McCune-Albright syndrome, Fanconi Anemia Tx : antibacterial soap, oral abx Carney Complex Etiology : inactivating mutation in PRKAR1A Inheritance : AD Appearance : hyperpigmented macules Location : labial, perioral, periorbital, anogenital Ass. with : cardiac myxoma, skin myxomas, lentiginosis, pituitary adenomas, testicular tumors, primary pigmented nodular adrenocortical disease 1/7 Carrión Disease (Verruga peruana / Peruvian wart) Etiology : sandflies that carry Bartonella bacilliformis At risk : South Americans Appearance : eruption of red to purple nodules Other sx : low grade fever, fatigue, headache, jaundice, pallor, HSM Tx : abx Cat Scratch Disease Etiology : cat carrying Bartonella henselae Appearance : erythematous papules + nodules with regional lymphadenopathy Other sx : fever, fatigue, headache, N/V, sore throat At risk : owning cats <12 mo old, licked/bitten/scratched by cat, immunocompromised Tx : self limiting or azithromycin for severe/persistent sx Cellulitis Etiology : S. pyogenes , S. aureus (often from a break in skin from trauma, infix , or recent surgery) Appearance : poorly-demarcated erythematous edematous plaque; typically unilateral At risk : middle age + older Location : lower extremities Tx : oral cephalexin or IV cefazolin Central Centrifugal Cicatricial Alopecia (CCCA) ***MC scarring hair loss Etiology : unknown, multifactorial At risk : African American females Location : vertex, frontal hair line Appearance : shiny scalp with follicular dropout Common sx : itchy scalp, burning sensation Tx : topical or intralesional CS, tacrolimus, Doxy Chancroid Etiology : STI transmission of H. ducreyi Appearance : one or more erythematous papules that quickly evolve into pustules and become larger until they break down into an ulcer Sx : extremely painful ulcer that bleeds easily, painful swollen lymph nodes in inguinal area Tx : azithromycin, ciprofloxacin, ceftriaxone or erythromycin 1/4 Cherry Angioma Etiology : aging; sometimes associated with somatic missense mutations in GNAQ and GNA11 (Q209H) genes Appearance : erythematous to blue or purple papule or nodule Location : trunk Tx : ED, laser, cryo Chickenpox Etiology : VZV Appearance : erythematous papules + vesicles in different stages (crust vs vesicle) Location : begins on trunk and spreads to face + extremities Tx : in children is self limited; in older children + adults acyclovir is recommended; VZIG for pregnant women Chillblains Etiology : tender and/or itchy bumps following exposure to damp, cold, non-freezing conditions causing constriction of small arteries and veins but a protective reflex intermittently dilates At risk : young to middle-aged adults, females Location : hands, feet, ears Tx : avoid cold, wet temps, topical nitroglycerine Prog : spontaneously regress in 1-3 weeks Chloasma / Melasma Etiology : overproduction of melanin by melanocytes Triggers: sun exposure, hormones, medications, ass. with family hx Appearance : light-to-dark brown macules or patches with irregular borders Location : bilateral cheeks At risk : pregnancy, females Tx : hydroquinone , tretinoin Chromoblastomycosis Etiology : chronic fungal infection; #1 fungus = Fonsecaea pedrosoi Appearance : papules + plaque that spread to surrounding tissue with a cauliflower appearance Location : limbs At risk : tropics; middle aged men Associated with : SCC Tx : itraconazole +/- terbinafine Coccidiomycosis / Valley Fever Etiology : allergic reaction to Coccidioides immitis Appearance : solitary or multiple indurated papules, nodules, pustules, abscesses, ulcers, and scars Reactive manifestation: erythema nodosum, erythema multiform e Ass. sx : low grade fever, chills, fatigue, cough, chest pain, joint pain, lymphadenopathy At risk : SW US, Central + South America, immunocompromised, farmers, construction workers, men Tx : self limiting (mild), fluconazole or itraconazole (moderate), or posaconazole or ampB (severe) 1/5 Collodion baby Define : newborn whose entire body is covered in tight, translucent membrane that resembles collodion Inheritance : AR Appearance : shiny, parchment-like membrane Tx : high-humidity incubator, emollients Comedones: Open + Closed Etiology : cells lining the sebaceous duct proliferate and there is increased sebum production causing debris blockage of the sebaceous duct and hair follicle Appearance : - open : gray, brown, black papules; keratinous contents that can be expressed - closed : skin colored papules Tx : benzoyl peroxide, azelaic acid, salicylic acid, tretinoin, adapelene Closed comedones Closed comedones Closed comedones Closed comedones Open comedones Open comedones Open comedones Condyloma Accuminata / Anogenital warts Etiology : low risk HPV strains (6,11) Appearance : soft tan-colored, cauliflower-like papules; appear 3-6 mo after infection Location : anus or genitals At risk : 15-30 yo, immunosuppressed Tx : cryo, podophyllin resin, trichloroacetic acid, electrosurgery Prevention : HPV vaccination Congenital Dermal Melanocytosis / Mongolian spots Etiology : entrapment of melanocytes in the dermis of developing embryo Appearance : blue-grey patches At risk : East Asians, Polynesians Location : shoulder, gluteal regions Prog : typically regress by puberty Tx : none Conradi-Hünermann-Happle syndrome Characterized by : skeletal abnormalities, skin lesions following Blaschko's lines, cataracts Inheritance : XLD (females >) Appearance : linear or whorled hyperkeratotic scales following the lines of Blaschko, follicular atrophoderma, pigmentary changes, and sometimes pustular lesions Cowden Syndrome Etiology : LOF mutation in PTEN Inheritance : AD Appearance : skin colored to yellow-brown, flat topped warty papules Ass. sx/conditions : autism, macrocephaly, thyroid goiter, breast cancer, GI polyps Location : central face surrounding eyes, nose, mouth Tx : 5-FU, oral retinoids To play, press and hold the enter key. To stop, release the enter key. Crusted Scabies / Norwegian Scabies Etiology : Sarcoptes scabiei var hominis Appearance : poorly defined erythematous patches that develop into thick scaly plaques At risk : immunocompromised, elderly, disabled or debilitated, HIV patients Location : between the fingers, under the nails, or diffusely over palms and soles, knees, and elbows Tx : oral ivermectin, topical insecticides Cushing Disease Etiology : prolonged exposure to cortisol = decreased collagen synthesis = BV rupture easier Appearance : purple striae (stretch marks), telangectasias, acne, hirsutism Location : abdomen Tx : remove inciting factor for increased cortisol Cutaneous Anthrax Etiology : Bacillus anthracis (spores inhaled in sheep and cattle, and then humans either get inoculated with it through minor cut or inhaled or ingested spores into lung) Appearance : papule with surrounding vesicles (develops after 1-7 days of exposure) that progress to an ulcer with black eschar and then heals into a scar within weeks Sx : painless At risk : farmers in Africa, Middle East , and Caribbean, workers in wool, hair, or bristle industries Tx : doxy (for uncomplicated cases), IV or IM penicillin (for traditional cases) at diagnosis 6 days after dx 4 weeks after dx Cutaneous Horn Etiology : underlying lesions are seborrheic keratosis, viral warts (due to HPV), actinic keratosis, or well-differentiated SCC (50/50 benign vs premalignant or malignant) Appearance : straight or curved, hard, yellow-brown projection from the skin At risk : 60+ yo, Fitzpatrick types 1/2 Location : sun-exposed areas Tx : excise depending on nature of lesion; always biopsy to R/O SCC Go to link Go to link Go to link Go to link Go to link Go to link Go to link Go to link Go to link 1/3 Cutaneous Larva Migrans Etiology : A. duodenale + N. americanus Appearance : erythematous serpiginous plaque; 2-3 mm-wide snakelike tracks stretching 3–4 cm from the penetration site Sx : burning or tingling sensation when worm penetrates and extreme itching when worm travels At risk : barefoot on the beach, children in sandpits, farmers Tx : self-limiting (hookworms will die on their own in 4-8 weeks) or can prescribe thiabendazole Cutaneous Leishmaniasis Etiology : Leishmania Appearance : initial lesion appears 2 weeks - 2 mo after sandfly bite and is a small red papule, which gradually enlarges up to 2 cm in diameter and forms an ulcerated nodule with raised border (volcano sign) Location : exposed skin, esp. face + extremities At risk : living or traveling through areas where sandflies and Leishmania species are endemic (Middle East, North Africa, Asia, Central and South America) Tx : self healing, topical non-antimonial (cryo, heat therapy, imiquimod), intralesional antimonial (sodium sibogluconate) Cutaneous Neurofibroma Etiology : unknown (solitary) or can be caused by NF1 gene mutation (multiple) Inheritance : AD Appearance : circumscribed, soft button-like brown, pink, or skin colored nodules with a soft or firm consistency; buttonhole sign Location : trunk Tx : no cure; excision for one or selumetinib may offer hope in reducing the size of plexiform neurofibromas 1/13 Cutis marmorata Etiology : superficial blood vessels dilate and contract at the same time Appearance : pinkish blue mottled or marbled appearance when subjected to cold temperatures At risk : children Prog : improves with age Cylindroma Etiology : mutation of CYLD gene that forms benign tumors of eccrine sweat glands Inheritance : AD Appearance : firm, rubbery, pink to bluish plaques and nodules Cysticercosis Etiology : Taenia solium (from ingestion of eggs in contaminated pork) Appearance : soft to firm, skin-colored nodules Ass. condition : seizures if involve brain (neurocysticercosis) Sx : painful Location : trunk + extremities Tx : praziquantel and albendazole A B C D E F G H I J K L M N O P Q R S T U V W X Y Z

Explore Daly’s Diverse Dermatology Dictionary: an inclusive resource with dermatology images on a range of skin tones for better, equitable medical education. Welcome to: Daly's Diverse Dermatology Dictionary By Letter... A H O V B I P W C J Q X D K R Y E L S F M T G N U Z Welcome! I’m so grateful you’re here. This resource was created with one goal in mind: to help bring greater diversity, accuracy, and inclusivity to dermatologic education. From my very first year in medical school, I noticed a glaring omission in our training, most dermatology images we learned from depicted conditions only on white skin. When darker skin tones were included, it was often only for diseases commonly associated with those populations, such as lupus or sarcoidosis. I searched everywhere for a comprehensive, side-by-side visual guide showing both common and uncommon conditions across the full range of skin tones… but that resource simply did not exist. What started as a personal frustration quickly turned into a mission. Throughout medical school, I began collecting and organizing photographs of dermatologic conditions in patients of all skin tones, building a databank to deepen my own learning. When classmates saw and used these materials, the response was immediate gratitude, surprise that such a tool wasn’t standard, and encouragement to make it something bigger. After hundreds of hours and countless images, I’m proud to share this passion project publicly: Bridging the Gap: Daly’s Diverse Dermatological Dictionary . It is not a fully exhaustive dermatology textbook, but rather a concise, essential, and intentionally curated resource highlighting the facts, visuals, and patterns I find most important, especially for medical students and early healthcare trainees. My only goal is to support better recognition of skin disease in EVERY patient. I have no financial disclosures, no sponsors, and no conflicts of interest. This work is created purely out of a commitment to more equitable, accurate medical education. Your enthusiasm fuels my own, and I’m honored to share this growing project with you. Important Note: Sensitive Content This educational resource includes clinical photographs of sensitive areas, including genitalia, when medically relevant. Viewer discretion is advised. Educational Purpose & Legal Disclaimer This dictionary is intended solely for educational purposes. It is NOT designed or approved for diagnosing, treating, or managing any medical conditions in real patients. Nothing in this document should be interpreted as medical advice, a treatment recommendation, or a substitute for professional clinical judgment. By accessing or viewing this dictionary, you acknowledge and agree that: You understand this resource is for educational enrichment only. You will not use any content within this document to diagnose or treat patients. The author (Paige Daly), DalyDerm, and all affiliated platforms assume no responsibility for clinical decisions made based on this material. All information is presented “as is” and without any warranty regarding accuracy, completeness, or fitness for a particular purpose. Dermatology is a constantly evolving field; details may change with new research, and occasional errors or omissions may occur despite best efforts. You agree to use this resource ethically, responsibly, and within your scope of training. If you believe something here is incorrect or have concerns about a specific entry, please reach out to me at dalydermatology@gmail.com so I can review and update the material accordingly. The author assumes no liability for any direct, indirect, incidental, or consequential damages arising from the use, or misuse, of this dictionary. By continuing to use this dictionary, you accept these terms fully. Welcome to a more inclusive way of learning dermatology. Thank you for being part of the movement to bridge the gap, together. Please enjoy, share widely, and if you’d like a downloadable version, scroll to the bottom. Your enthusiasm fuels my own, and I’m grateful to share this journey with you. — Paige O’Brien Daly, MA Creator of DalyDerm & Dermatology Educator Submit Your Own Photo Please take a moment to fill out the form. First Name Last Name Email Diagnosis Leave us a message and include: (1) that you consent to allowing me to use your photo within my dictionary (2) the photo and diagnosis. Submit Thanks for submitting!

I have only linked produ cts that I have tried & liked! Most up to date items are on my LinkTree Account, go check it out!! Nothing is sponsored. All items with a red heart on it are what I am currently using. <3 LinkTree Account Cleansers You get the most out of cleansers when consistently use a non-exfoliating cleanser it in the AM and PM to remove dirt, oils and more that you have built up throughout the day or night. I like to use an exfoliating facial cleanser 2-3 x a week to remove excess dead skin! I also prefer a hydrating or "non-foaming" cleanser because it personally strips my skin barrier. Serums After using a cleanser, I have found that serums (Vitamin C for AM, retinol for PM) are best to use right after and then use lotions to follow and seal in all the serums you applied. For the Body Facial Lotions Sunscreen Hands down the most important part of your skincare routine! Yes, I am naturally fair-skinned, but sunscreen is not only good at preventing skin cancer, but preventing wrinkles and improving overall skin tone. For Keratosis Pilaris I do not naturally have the smoothest skin and am still working on bettering the skin on my body. What I have found that works for me is consistent exfoliation and lotions during the AM & PM. I am always looking for recommendations too! :)

Hi! I’m Paige, an OMS-4 student. Welcome to Daly Dermatology: Your guide to growing your confidence and knowledge about your skin while getting tips on work-life balance from a current medical student! I was diagnosed with vitiligo at a young age and have struggled with other skin conditions my whole life and therefore struggled with confidence. I am still growing in my confidence journey, but by expanding my knowledge of how to better my own skin, I am becoming more confident in who I am. I am excited to share with you what I have learned and experienced while on a budget (because you know, med school) and I am excited for you to join me on my journey while I root for you on yours! So welcome! I am so excited! Student Dr. Paige Daly To play, press and hold the enter key. To stop, release the enter key.

All Posts By: Paige All Posts Science in Skincare Medical School New Products I Have Tried Fun Facts! Search Paige Daly 5 minutes ago 5 min How I Passed COMLEX 1 and Step 1 AND Got Married in the Same Month With careful planning and dedication, balancing medical school, board exams, and personal life is not just possible—it's achievable! Post not marked as liked Paige Daly Feb 7 3 min Unlocking the Science of Skincare: A Medical Student's Journey into Active Ingredients As a medical student, my days are filled with lectures, labs, and countless hours of studying. Amidst the chaos of medical school, I find... Post not marked as liked Paige Daly Dec 12, 2023 2 min The Science Behind Pimple Patches: Uncovering the Secrets to Clear Skin Pimples – the bane of every teenager's existence. Whether you're battling occasional breakouts or dealing with persistent acne, the quest... Post not marked as liked Paige Daly Dec 8, 2023 2 min My Illuminating Discovery with DRMTLGY's Brightening Eye Masks In the ever-evolving world of skincare, one brand recently caught my attention and became part of my self-care routine: DRMTLGY.... Post not marked as liked 1 2 3 4 Paige Daly Welcome to DalyDermatology! 💖 Dive into the world of skincare science with me as we explore the secrets behind that radiant glow. Join me on this journey as a medical student, continually learning and sharing my experiences in dermatology. ✨ follow me @dalyderm