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Bacterial Folliculitis
Etiology: S. aureus, unless it is Gram negative (Escherichia coli, Pseudomonas aeruginosa, Serratia marcescens, Klebsiella or Proteus species) or Hot tub Folliculitis (Pseudomonas aeruginosa)
Appearance: follicular pustules, erythematous nodules
Tx: warm compress, anti-inflammatories, mupirocin
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Behçet Disease
Etiology: unknown – assumed to be connected to autoimmune response
Appearance: oral + genital ulcers that are 3-5 mm, round to oval ulcers with peripheral rim of erythema + yellowish adherent exudate centrally (indistinguishable from aphthous ulcers)
At risk: eastern + central Asian; ~35 yo
Blastomycosis
Etiology: Blastomyces dermatitidis
Appearance: purplish-gray verrucous lesions with heaped borders or friable lesions that ulcerate
Location: face, neck, extremities
Tx: itraconazole, amp B




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Bowenoid papulosis
Etiology: HPV 16,18
Appearance: reddish-brown papules
Location: anogenital region
Tx: ED, laser, cryo




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Bullous Diabeticorum
Etiology: diabetes
Location: lower extremities
Tx: self resolving




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Bullous Impetigo
Etiology: S. aureus (exfoliative toxins A + B which targets desmoglein 1)
Appearance: thin roofed bullae that tend to rupture spontaneously and ooze leaving a yellow crusty rim
At risk: <2 yo
Location: face, trunk, extremities, buttocks, perineal regions
Tx: oral flucloxacillin
Burns
Etiology: any external heat/radiation source
Types:
- Superficial burn = localized, dry, blanching redness with no blisters
- Superficial partial-thickness burn = blisters, blanches with pressure, swollen, warm
- Deep partial-thickness burn = blisters that are easily unroofed, does not blanch with pressure and painful only to deep pressure
- Full-thickness burn = white, waxy, dry, inelastic, leathery, does not blanch with pressure, painless
- Deeper injury burn = white, dry, inelastic, does not blanch with pressure, painless
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