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H Hailey Hailey Etiology: Halo nevus Etiology : unknown, but suspected to related to autoimmune in origin and triggered by sunburn or local trauma Description : white halo that surrounds a mole/nevus and is usually symmetrical and about 0.5–1.0 cm wide Location : most often trunk Hand Foot & Mouth Disease Etiology : Coxsackie A Description : vesicular rash Location : around the lips, mouth, and back of the throat Go to link Go to link Go to link Go to link Go to link Go to link Go to link Go to link Go to link 1/2 Hand-Foot Syndrome Etiology : chemotherapy agents (5-FU, capecitabine) Description : sunburnt-like hands that appears 2-3 months after starting chemotherapy where the whole sole or palm turns completely red 1/6 Hand-Schüller-Christian Disease Etiology : unknown cause, but the number of Langerhans cells in the skin and other organs are triggered to increase (type of Langerhans cell histiocytosis); BRAF V600E mutation associated Description : Pinkish crusted papules with lytic bone lesions in skull, diabetes insipidus, and exophthalmos At risk : 2-6 yo 1/2 Harlequin Ichthyosis Define : severe genetic skin disorder characterized by thick, diamond-shaped plates of skin separated by deep cracks Tx : oral retinoids Hereditary Hemorrhagic Telangiectasia / Osler-Weber-Rendu Etiology : AD mutation (loss of function of ENG gene) causing thin-walled blood vessels, especially in the mouth and GI tract Description : blanching lesions (telangiectasias) on skin and mucous membranes, recurrent epistaxis, skin discolorations, arteriovenous malformations, GI bleeding, hematuria Herpes Labialis (cold sores) Etiology : HSV1 Description : shallow oral ulcers that rupture and dry after a few days forming a thin, yellowish crust and will completely heal At risk : children (transmitted through contact with oral secretions) Herpes Zoster / Shingles Etiology : recurrent VZV Description : eruption of a “belt” of maculopapular lesions with erythematous base along a single dermatome on the trunk To play, press and hold the enter key. To stop, release the enter key. Hidradenitis Suppurativa (HS) / Acne Inversa Etiology: occlusion + rupture of follicles in folliculopilosebaceous units Locations : axillae, groin, perineum, perianal, inframammary At risk : puberty, women, obesity, cigarette smoking, IBD Appearance : erythematous nodules, pustules, bridging comedones Sx : recurrent tender nodules + abscesses that drain + scar Tx : mild = Hibiclens (1 min soak + then wash), Doxy, Clinda lotion, spironolactone severe/scarring = biologic (Bimzelx, Humira) Hirsutism Etiology: late-onset CAH, prolactinoma, drugs (androgens, minoxidil, cyclosporine, carbamazepine) Location : mustache or beard-liike distribution Tx : correcting underlying hormonal imbalance (spironolactone, OCPs) + hair removing tx Hypertrichosis Description : excessive hair growth Associated with : Porphyria cutanea tarda, anorexia nervosa, malignancy, drugs Hypertrophic Scar Etiology : dysregulation of the normal healing process resulting in excessive production of collagen, elastin, proteoglycans, and extracellular matrix proteins Description : Pink to red, slightly raised, or flat, limited to confines of wound, and usually occurs within weeks of injury Hypohidrotic Ectodermal Dysplasia Define : genetic disorder characterized by the abnormal development of ectodermal tissues, specifically the skin, hair, nails, teeth, and sweat glands Appearance : thin, wrinkled, and dark-colored skin around the eyes A B C D E F G H I J K L M N O P Q R S T U V W X Y Z

F Female Pattern Hair Loss Etiology: hereditary Appearance : widening of hair part; thinning, no shining of scalp Location : mid-frontal scalp Tx : topical minoxidil, oral spironolactone Fibrous Hamartoma of Infancy Etiology: benign tumor of the subcutis and lower dermis Histo: "ramen" waves - grey/blue ball cells = immature mesenchyme - mature fat that is honeycomb like At risk : < 2 yo Tx : excision Fixed Drug Eruption Etiology: drugs (antimicrobials and NSAIDs are most common); delayed type IV hypersensitivity reaction Appearance: - Early lesions: sharply demarcated erythematous macules - Late lesions: round or oval plaques that may evolve to become bullae and then erosions (occur 30 - 8 hours after ingesting drug) Location: mouth, genitalia, face, hands, feet Prog: resolve days to weeks after drug is discontinued; PIH may persist Tx : discontinue suspected medication, topical or systemic CS Folliculitis Decalvans Etiology: considered to be the result of an abnormal immune response to S. aureus , although this is not yet proven Appearance: erythematous, swollen, and scaly patches and plaque with crust and pustules (like CCCA, but smaller and with pustules) Sx: itching, pain, and burning sensation Tx: clindamycin, CCS (topical + oral or injected) Focal Dermal Hypoplasia / Goltz Syndrome Etiology: rare genetic disorder affecting tissues derived from ectoderm and mesoderm Inheritance : XLD Location : mouth, genitals, anus Ass. conditions : coloboma, missing or webbed fingers, hypoplastic teeth, low set ears, cleft lip or palate, bladder exstrophy, kidney abnormalities, CHD, vision and hearing impairment Appearance : thin or absent skin areas, hypopigmentation, hyperpigmentation, and fat herniation; following Blaschko's lines; wart-like growth Tx : vascular lasers for cutaneous papillomas and telangectatic areas Frey's Syndrome Etiology: postoperative complication involving parasympathetic fibers regenerating on sympathetic fibers of sweat glands (auriculotemporal branch of V3 is cut during an excision of the parotid gland and reinnervates the skin sweat glands in front of the ear) Appearance : redness + sweating Location : preauricular Triggers : post-eating, thinking or dreaming about food Tx : symptom control, botox, aluminum antiperspirant, oral or topical glycopyrrolate 1/2 A B C D E F G H I J K L M N O P Q R S T U V W X Y Z

I have only linked produ cts that I have tried & liked! Most up to date items are on my LinkTree Account, go check it out!! Nothing is sponsored. All items with a red heart on it are what I am currently using. <3 LinkTree Account Cleansers You get the most out of cleansers when consistently use a non-exfoliating cleanser it in the AM and PM to remove dirt, oils and more that you have built up throughout the day or night. I like to use an exfoliating facial cleanser 2-3 x a week to remove excess dead skin! I also prefer a hydrating or "non-foaming" cleanser because it personally strips my skin barrier. Serums After using a cleanser, I have found that serums (Vitamin C for AM, retinol for PM) are best to use right after and then use lotions to follow and seal in all the serums you applied. For the Body Facial Lotions Sunscreen Hands down the most important part of your skincare routine! Yes, I am naturally fair-skinned, but sunscreen is not only good at preventing skin cancer, but preventing wrinkles and improving overall skin tone. For Keratosis Pilaris I do not naturally have the smoothest skin and am still working on bettering the skin on my body. What I have found that works for me is consistent exfoliation and lotions during the AM & PM. I am always looking for recommendations too! :)

Y No conditions to list. A B C D E F G H I J K L M N O P Q R S T U V W X Y Z

I Ichthyosis vulgaris Etiology : loss-of-function mutations in the gene encoding the protein filaggrin Description : Dry scaly skin; most often it appears after about 2 months Location : extensor side of extremities, scalp, central face, and trunk Immunoglobulin A vasculitis / Henoch-Schönlein Purpura Etiology : Small vessel vasculitis Description : palpable purpura Location : buttocks + legs Bullous HSP Classic Purpura Classical purpura Ankle swelling (arthritis) Henoch-Schönlein purpura Henoch-Schönlein purpura Close-up of purpuric rash with vesicle (blister) formation Henoch-Schönlein purpura Impetigo Etiology: S. aureus > S. pyogenes Appearance : thin-walled vesiculopustules that quickly rupture; exudate with honey color and dried crust At risk : 2-5 yo, contact sports, broken skin, humid weather Location : face > extremities Tx : bacitracin, mupirocin Incontinentia pigmenti Etiology : X-linked dominant disorder Description : 4 stages of lesions: vesicular (red, blister-like lesions), verrucal (wart-like lesions), hyperpigmentation and hypopigmentation in a marble like pattern Infantile Acne Etiology: 1/5 Intradermal Nevus Etiology : acquired nevus where melanocytes are found in the dermal layer of skin Description : raised, dome shaped papules less than 10mm in diameter Intertrigo Appearance : satellite macules, papules or pustules around the erythema in the fold Sx : burns more than itches Location : inframammary fold, gluteal cleft, inguinal creases, folds under pannus Tx : clotrimazole cream 1/12 Irritant Contact Dermatitis (ICD) Etiology : physical and chemical irritants (wood, dust, soaps) Description : burning and painful sensation with red patches To play, press and hold the enter key. To stop, release the enter key. A B C D E F G H I J K L M N O P Q R S T U V W X Y Z

Welcome Welcome Welcome Welcome Welcome to DalyDermatology! 💖 Join me on my journey as a medical student exploring the science behind healthy, radiant skin—and working to make dermatology more inclusive for everyone. Inspired by the lack of diverse skin images in medical education, I created Daly’s Diverse Dermatology Dictionary: a free resource showing how skin conditions appear across all skin tones. My passion is to bridge gaps in knowledge, encourage equity, and help our medical community better serve every patient. Thanks for being here—let’s learn and grow together! ✨ Check out my most recent blogs! Let Me Catch You Up 🤍 The past few months have been full: audition rotations, applications, interviews, and quiet growth behind the scenes. In this post, I’m catching you up on the reality of dermatology and TY interview season, what this stretch has taught me, and why sometimes the quiet seasons are the ones building the strongest foundation. Step 2 CK Exam Day: What I Wear, What I Pack, and How I Stay Focused Wondering what to wear, pack, and do on Step 2 CK exam day? I break down my morning routine, must-have snacks, bag essentials, and in-exam strategies for staying sharp and confident. Get my real-life tips for managing nerves and maximizing performance on test day! Derm Dream Bag: My Dermatology Audition Rotation Essentials Discover the ultimate dermatology audition bag essentials! From pink Vaseline and L’Oréal lip gloss to study guides, sunscreen, meds, and snacks—this tote is packed for a busy med student’s clinic day. Stay prepped, polished, and confident with these must-haves in your Poppy & Peonies tote. Bridging the Gap: Daly's Diverse Dermatology Dictionary After realizing most dermatology images in my training only showed conditions on white skin, I began collecting and organizing photos of skin diseases across all skin tones. This project grew into Daly’s Diverse Dermatology Dictionary—a free, ever-expanding resource designed to fill this gap and help students, clinicians, and educators recognize skin diseases on every patient. My goal is to make dermatology education more inclusive, accurate, and truly reflective of the diversity in our world, so we can provide better, more equitable care for all. Click Here Follow me on Instagram @dalyderm Load more

D Darier Disease / Keratosis Follicularis Etiology: mutation of ATP2A2 gene Inheritance : AD Appearance : scaly crusted papules and greasy plaque; alternating red + white nail beds with V-shaped nicking Location : seborrheic dermatitis distribution (oily areas of the body like chest, back, scalp margins, forehead, nasolabial folds, eyebrows, beard) and skin folds At risk : adolescents Tx : moisturizers, topical retinoids, sun protection Dermatitis Herpetiformis Etiology: cross-reactivity between anti-gliadin IgA antibodies + transglutaminase at the dermal basement membrane Appearance : papulovesicles or excoriated papules on erythematous base Location : elbows, dorsal forearms, knees, scalp, buttocks Sx : intense itching At risk : pts with Celiac Disease, Irish or Swedish patients Associated with : maltomas Tx : gluten free diet, dapsone Dermatofibroma Etiology : trauma (mosquito bite, shaving nick) Appearance : tan to pink, firm, hyperpigmented dome-shaped papules with peripheral rim of darkening pigment Location : extremities Prog : once you get one, you're at risk of getting more Dermoscopy : peripheral fine network, central white or pink scar-like area, ring-like globules, dotted vessels At risk : adults Test : "dimple or pinch" sign Tx : reassurance Dermatofibrosarcoma protuberans Etiology : rare type of skin cancer characterized by its slow-growing, locally aggressive nature; unknown cause, but an injury is a predisposing factor Appearance : skin colored, pink or brown irregular border multi nodular fungating mass Histo : storiform spindle cells very deep with fat getting trapped in it Sx : painless plaque +/- nodules that feels rubbery or firm to touch At risk : adults between 20- 60 yo Location : trunk Tx : MOHs favored over wide excision; imatinib if t17;22 To play, press and hold the enter key. To stop, release the enter key. Dermatographia Etiology : skin trauma may release an antigen that reacts with the membrane bound IgE on mast cells triggering histamine release Appearance : linear wheals + a surrounding erythematous flare which appears 1-3 minutes after stroking + resolves in 30-60 minutes At risk : young adults Tx : loose fitting clothes, avoid triggers, antihistamines, phototherapy, omalizumab 1/7 Dermatomyositis Etiology : unknown; classic model considers DM to be the result of a humoral attack against the endothelium of muscle capillaries + small arterioles; risk factors include underlying malignancy + family hx of autoimmune disease At risk : 30-50 yo, females Sx : rash appears weeks to months before proximal muscle weakness Locations : extensor surfaces of MCP + IP = Gottron papules upper eyelids = Heliotrope malar-like rash chest = V neck sign = Poikiloderma upper back = Shawl sign lateral thigh = Holster sign Ass. conditions : adenocarcinomas Tx : systemic CS Go to link Go to link Go to link Go to link Go to link Go to link Go to link Go to link Go to link 1/3 Dermatophytosis / Tinea / Ringworm Etiology: Trichophyton, Epidermophyton, Microsporum Appearance : annular erythematous plaque with central clearing and raised scaly edge Sx : itchiness Locations : tinea barbae = beard tine a c apitis = head tinea corporas = body tinea cruris = groin tinia unguium = nails tine a pedis = foot At risk : hot humid climates Tx : topical antifungals (imidazole, terbinafine) unless if it is on head or nails (oral instead: itraconazole, itraconazole) Dermatosis Papulosa Nigra Etiology : activating mutation in FGFR3 Appearance : 1-5 mm in diameter, hyperpigmented brown-black sessile to filiform, smooth surfaced papules Location : cheeks, temples At risk : Fitzpatrick types 4/5/6, females Tx : EDC (for darker skin), cryo (for lighter skin), Nd:YAG laser 1/10 Diffuse Palmoplantar Keratoderma (DPK) Etiology: genetic abnormality Inheritance : AR or AD Appearance : hyperkeratosis with white/yellow hue Sx : hyperhidrosis Location : symmetric palmar + plantar surfaces Tx : emollients, keratolytic agents ( salicylic acid, propylene glycol), topical retinoids, oral retinoids (acitretin), topical vitamin D (calcipotriol) Disseminated Superficial Actinic Porokeratosis (DSAP) Etiology: mutation in: MVD, MVK, FDPS, PMVK or SART3 genes; results in decreased cholesterol in the affected areas of the skin Inheritance : AD At risk : European, females Locations : arms, legs Appearance : irregular annular plaque with elevated horny rim Prog : <10% turn into SCC Tx : compounded off-label topical 2% lovastatin +/- topical cholesterol, sun protection education Drug Reaction with Eosinophilia and Systemic Symptoms (DRESS) Etiology: delayed type IV HS reaction to certain medication ( anti epileptics , allopurinol, sulfonamides, minocycline, HIV meds) Appearance : facial edema, diffuse erythematous macules + plaques that typically occur in 3rd week after starting a med or increasing dose Ass. sx : high fever, lymphadenopathy, hematological abnormalities, hepatitis, nephritis, carditis, other organ involvement, facial swelling Location : first involves face, upper trunk, UE, and then spreads to LE Tx : stop all suspect medications, systemic CCS, cyclosporin, IVIG Dyshidrotic Eczema Etiology : unknown Location: palms, fingers, soles of feet Sx : extremely itchy chronic, recurrent Appearance : recurrent fluid-filled vesicles that resolve after several weeks with scaling Tx : clobetasol + tacrolimus; severe = Dupixent Dyskeratosis Congenita (DC) Etiology : 14 different genes (DKC1 gene mutations on X chromosome); causing telomere shortening = premature aging Inheritance : AD or AR Appearance : lacy reticular hyperpigmentation Other sx : nail dystrophy, oral leukoplakia, early hair greying, sparse eyelashes, hyderhidrosis Location : upper chest, neck, nail atrophy, oral leukoplakia Tx : no cure; tx is aimed at maintaining bone marrow function via oxymetholone as this is the major cause of death A B C D E F G H I J K L M N O P Q R S T U V W X Y Z

Z Zoon’s Balanitis Etiology: unknown, potentially from reactive response to an infection, injury, or irritation Histo : plasma cells Appearance: red to orang, shiny plaque Location: glans penis Tx : topical CS, A B C D E F G H I J K L M N O P Q R S T U V W X Y Z

Q No conditions to list. A B C D E F G H I J K L M N O P Q R S T U V W X Y Z

W Waardenburg Syndrome Etiology: mutations in genes involving the development of melanocytes + neural crest cells Inheritance : AD Appearance : patchy depigmentation of skin, hair Ass. conditions : deafness, heterochromia iridis Tx : no cure, sun protection, audiology exam Warfarin-induced skin necrosis Etiology: warfarin decreases protein C = poor inactivation of factor Va and VIIIa = hyper coagulable state Timing : first 2-5 days of starting warfarin Appearance : purport that becomes bluish-black with a red rim over a few days; blood blisters and full thickness skin necrosis follows Locations : extremities, breasts, trunk, penis Ass. conditions : protein C deficiency At risk: 50-70 yo, obese pts, perimenopausal women Tx : stop warfarin, Vitamin K reverses warfarin quicker, may require surgery or skin grafting Waterhouse-Friderichsen Syndrome (meningococcal disease) Etiology: Meningococcus which leads to adrenal hemorrhage, fulminant sepsis, and DIC Appearance : petechial or purpura that do not blanch Ass. sx : neck stiffness, HA, N/V, fevers, chills, irritability, seizure Location : begins on trunk or legs At risk : less than 4 yo, complement deficiencies, asplenic patients Tx : Penicillin, 3rd gen cephalosporin, IVF, NE, platelets Prog : if not treated quickly, pt may rapidly die A B C D E F G H I J K L M N O P Q R S T U V W X Y Z

U Urticaria / Hives Etiology: mast cell degranulation from allergic reaction (T1HS) or AutoAb to FcER1 (T2HS) Appearance: wheals that disappear within 24 hours; surrounding erythema will blanch Ass. conditions : angioedema Sx : pruritus Tx : avoid trigger, antihistamines (cetirizine, loratadine ), omalizumab, cyclosporin Urticarial vasculitis Etiology: rare inflammatory condition affecting small blood vessels in the skin Ass. with : SLE, Sjogrens, leukemia, viral conditions, drug induced Sx : painful or burning sensation Appearance : wheals that will last longer than 24 hours Prog : will go away in 1-3 years of onset Tx : antihistamines, NSAIDs, CCS, immunosuppressant, omalizumab, dapsone A B C D E F G H I J K L M N O P Q R S T U V W X Y Z

Hi! I’m Paige, an OMS-4 student. Welcome to Daly Dermatology: Your guide to growing your confidence and knowledge about your skin while getting tips on work-life balance from a current medical student! I was diagnosed with vitiligo at a young age and have struggled with other skin conditions my whole life and therefore struggled with confidence. I am still growing in my confidence journey, but by expanding my knowledge of how to better my own skin, I am becoming more confident in who I am. I am excited to share with you what I have learned and experienced while on a budget (because you know, med school) and I am excited for you to join me on my journey while I root for you on yours! So welcome! I am so excited! Student Dr. Paige Daly To play, press and hold the enter key. To stop, release the enter key.